Biblio
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“Varying Degrees of Temporoparietal Hypometabolism on FDG-PET Reveal Amyloid-Positive Logopenic Primary Progressive Aphasia is not a Homogeneous Clinical Entity.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1019-1029, 2017.
, “Visit-to-Visit Blood Pressure Variability and Alzheimer's Disease: Links and Risks.”, J Alzheimers Dis, vol. 59, no. 2, pp. 515-526, 2017.
, “Visuospatial Functioning in Cerebral Amyloid Angiopathy: A Pilot Study.”, J Alzheimers Dis, vol. 56, no. 4, pp. 1223-1227, 2017.
, “Vitamin D and the Risk of Dementia: The Rotterdam Study.”, J Alzheimers Dis, vol. 60, no. 3, pp. 989-997, 2017.
, “What Drives Country Differences in Cost of Alzheimer's Disease? An Explanation from Resource Use in the GERAS Study.”, J Alzheimers Dis, vol. 57, no. 3, pp. 797-812, 2017.
, “Young to Middle-Aged Dogs with High Amyloid-β Levels in Cerebrospinal Fluid are Impaired on Learning in Standard Cognition tests.”, J Alzheimers Dis, vol. 56, no. 2, pp. 763-774, 2017.
, “Young to Middle-Aged Dogs with High Amyloid-β Levels in Cerebrospinal Fluid are Impaired on Learning in Standard Cognition tests.”, J Alzheimers Dis, vol. 56, no. 2, pp. 763-774, 2017.
, “Accurate Prediction of Conversion to Alzheimer's Disease using Imaging, Genetic, and Neuropsychological Biomarkers.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1143-59, 2016.
, “Activation of p53 in Down Syndrome and in the Ts65Dn Mouse Brain is Associated with a Pro-Apoptotic Phenotype.”, J Alzheimers Dis, vol. 52, no. 1, pp. 359-71, 2016.
, “Active Cigarette Smoking in Cognitively-Normal Elders and Probable Alzheimer's Disease is Associated with Elevated Cerebrospinal Fluid Oxidative Stress Biomarkers.”, J Alzheimers Dis, vol. 54, no. 1, pp. 99-107, 2016.
, “Activities of Daily Living and Depressive Symptoms in Patients with Subjective Cognitive Decline, Mild Cognitive Impairment, and Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1043-50, 2016.
, “Age-Dependent Regulation of the Blood-Brain Barrier Influx/Efflux Equilibrium of Amyloid-β Peptide in a Mouse Model of Alzheimer's Disease (3xTg-AD).”, J Alzheimers Dis, vol. 49, no. 2, pp. 287-300, 2016.
, “Age-Dependent Regulation of the Blood-Brain Barrier Influx/Efflux Equilibrium of Amyloid-β Peptide in a Mouse Model of Alzheimer's Disease (3xTg-AD).”, J Alzheimers Dis, vol. 49, no. 2, pp. 287-300, 2016.
, “Age-Related Changes in the Synaptic Density of Amyloid-β Protein Precursor and Secretases in the Human Cerebral Cortex.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1209-14, 2016.
, “Alcohol Consumption and Incident Dementia: Evidence from the Sydney Memory and Ageing Study.”, J Alzheimers Dis, vol. 52, no. 2, pp. 529-38, 2016.
, “Altered Affective Evaluations of Smells in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 2, pp. 433-41, 2016.
, “Anemia and Mild Cognitive Impairment in the German General Population.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1031-42, 2016.
, “Anemia and Mild Cognitive Impairment in the German General Population.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1031-42, 2016.
, “Anemia and Mild Cognitive Impairment in the German General Population.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1031-42, 2016.
, “Anti-Correlated Cerebrospinal Fluid Biomarker Trajectories in Preclinical Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1085-97, 2016.
, “The Anti-Prion Antibody 15B3 Detects Toxic Amyloid-β Oligomers.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1485-97, 2016.
, “Anti-Viral Properties of Amyloid-β Peptides.”, J Alzheimers Dis, vol. 54, no. 3, pp. 859-878, 2016.
, “Apo J and Apo D: Complementary or Antagonistic Roles in Alzheimer's Disease?”, J Alzheimers Dis, vol. 53, no. 2, pp. 639-50, 2016.
, “Association of Butyrylcholinesterase-K Allele and Apolipoprotein E ɛ4 Allele with Cognitive Decline in Dementia with Lewy Bodies and Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 2, pp. 567-76, 2016.
, “Association of Physical Function with Clinical and Subclinical Brain Disease: The Framingham Offspring Study.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1597-608, 2016.
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