Biblio
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“Increased Intrinsic Activity of Medial-Temporal Lobe Subregions is Associated with Decreased Cortical Thickness of Medial-Parietal Areas in Patients with Alzheimer's Disease Dementia.”, J Alzheimers Dis, vol. 51, no. 1, pp. 313-26, 2016.
, “Inflammation, Antiinflammatory Agents, and Alzheimer's Disease: The Last 22 Years.”, J Alzheimers Dis, vol. 54, no. 3, pp. 853-857, 2016.
, “Influence of Incipient Dementia on Hospitalization for Primary Care Sensitive Conditions: A Population-Based Cohort Study.”, J Alzheimers Dis, vol. 52, no. 1, pp. 213-22, 2016.
, “Integrating Biomarkers for Underlying Alzheimer's Disease in Mild Cognitive Impairment in Daily Practice: Comparison of a Clinical Decision Support System with Individual Biomarkers.”, J Alzheimers Dis, vol. 50, no. 1, pp. 261-70, 2016.
, “JNK: A Putative Link Between Insulin Signaling and VGLUT1 in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 4, pp. 963-7, 2016.
, “JNK: A Putative Link Between Insulin Signaling and VGLUT1 in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 4, pp. 963-7, 2016.
, “The Language Profile of Behavioral Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 50, no. 2, pp. 359-71, 2016.
, “Late-Life Depressive Symptoms and Lifetime History of Major Depression: Cognitive Deficits are Largely Due to Incipient Dementia rather than Depression.”, J Alzheimers Dis, vol. 54, no. 1, pp. 185-99, 2016.
, “The Latent Dementia Phenotype δ is Associated with Cerebrospinal Fluid Biomarkers of Alzheimer's Disease and Predicts Conversion to Dementia in Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 49, no. 2, pp. 547-60, 2016.
, “The Latent Dementia Phenotype δ is Associated with Cerebrospinal Fluid Biomarkers of Alzheimer's Disease and Predicts Conversion to Dementia in Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 49, no. 2, pp. 547-60, 2016.
, “The Latent Dementia Phenotype δ is Associated with Cerebrospinal Fluid Biomarkers of Alzheimer's Disease and Predicts Conversion to Dementia in Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 49, no. 2, pp. 547-60, 2016.
, “Life's Simple 7's Cardiovascular Health Metrics are Associated with Hispanic/Latino Neurocognitive Function: HCHS/SOL Results.”, J Alzheimers Dis, vol. 53, no. 3, pp. 955-65, 2016.
, “Life's Simple 7's Cardiovascular Health Metrics are Associated with Hispanic/Latino Neurocognitive Function: HCHS/SOL Results.”, J Alzheimers Dis, vol. 53, no. 3, pp. 955-65, 2016.
, “Linguistic Features Identify Alzheimer's Disease in Narrative Speech.”, J Alzheimers Dis, vol. 49, no. 2, pp. 407-22, 2016.
, “Living Alone with Dementia: Prevalence, Correlates and the Utilization of Health and Nursing Care Services.”, J Alzheimers Dis, vol. 52, no. 2, pp. 619-29, 2016.
, “Longitudinal Alteration of Intrinsic Brain Activity in the Striatum in Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 54, no. 1, pp. 69-78, 2016.
, “The Longitudinal Association Between a Discrepancy Measure of Anosognosia in Patients with Dementia, Caregiver Burden and Depression.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1133-43, 2016.
, “Longitudinal Relationships between Caloric Expenditure and Gray Matter in the Cardiovascular Health Study.”, J Alzheimers Dis, vol. 52, no. 2, pp. 719-29, 2016.
, “Low Prevalence of Mixed Dementia in a Cohort of 2,000 Elderly Patients in a Memory Clinic Setting.”, J Alzheimers Dis, vol. 50, no. 3, pp. 797-806, 2016.
, “Low Serum Insulin-Like Growth Factor-I Predicts Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 2, pp. 641-9, 2016.
, “Lower Late-Life Body-Mass Index is Associated with Higher Cortical Amyloid Burden in Clinically Normal Elderly.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1097-105, 2016.
, “LRP/LR Antibody Mediated Rescuing of Amyloid-β-Induced Cytotoxicity is Dependent on PrPc in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 3, pp. 645-57, 2016.
, “Maintenance of Cognitive Performance and Mood for Individuals with Alzheimer's Disease Following Consumption of a Nutraceutical Formulation: A One-Year, Open-Label Study.”, J Alzheimers Dis, vol. 51, no. 4, pp. 991-5, 2016.
, “MAPT H1 Haplotype is Associated with Late-Onset Alzheimer's Disease Risk in APOEɛ4 Noncarriers: Results from the Dementia Genetics Spanish Consortium.”, J Alzheimers Dis, vol. 49, no. 2, pp. 343-52, 2016.
, “MAPT H1 Haplotype is Associated with Late-Onset Alzheimer's Disease Risk in APOEɛ4 Noncarriers: Results from the Dementia Genetics Spanish Consortium.”, J Alzheimers Dis, vol. 49, no. 2, pp. 343-52, 2016.
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