Biblio
“Association of Physical Function with Clinical and Subclinical Brain Disease: The Framingham Offspring Study.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1597-608, 2016.
, “Interaction Between Midlife Blood Glucose and APOE Genotype Predicts Later Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1553-62, 2016.
, “Genetic Interaction with Plasma Lipids on Alzheimer's Disease in the Framingham Heart Study.”, J Alzheimers Dis, vol. 66, no. 3, pp. 1275-1282, 2018.
, “Accelerometer-Measured, Habitual Physical Activity and Circulating Brain-Derived Neurotrophic Factor: A Cross-Sectional Study.”, J Alzheimers Dis, vol. 85, no. 2, pp. 805-814, 2021.
, “Association of Midlife Depressive Symptoms with Regional Amyloid-β and Tau in the Framingham Heart Study.”, J Alzheimers Dis, vol. 82, no. 1, pp. 249-260, 2021.
, “Plasma EFEMP1 Is Associated with Brain Aging and Dementia: The Framingham Heart Study.”, J Alzheimers Dis, vol. 85, no. 4, pp. 1657-1666, 2021.
, “Associations Between Brainstem Volume and Alzheimer's Disease Pathology in Middle-Aged Individuals of the Framingham Heart Study.”, J Alzheimers Dis, vol. 86, no. 4, pp. 1603-1609, 2022.
, “Associations of Pulmonary Function with MRI Brain Volumes: A Coordinated Multi-Study Analysis.”, J Alzheimers Dis, vol. 90, no. 3, pp. 1073-1083, 2022.
, “Insulin-Like Growth Factor, Inflammation, and MRI Markers of Alzheimer's Disease in Predominantly Middle-Aged Adults.”, J Alzheimers Dis, vol. 88, no. 1, pp. 311-32, 2022.
, “Non-Alcoholic Fatty Liver Disease, Liver Fibrosis, and Regional Amyloid-β and Tau Pathology in Middle-Aged Adults: The Framingham Study.”, J Alzheimers Dis, vol. 86, no. 3, pp. 1371-1383, 2022.
, “Associations of Physical Activity Engagement with Cerebral Amyloid-β and Tau from Midlife.”, J Alzheimers Dis, vol. 100, no. 3, pp. 935-943, 2024.
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