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Top 50Extracellular Tau SpreadingAlzheimer’s Funding AnalyzerBook and mansimvastatinplaquesstainingfractional_anisotropy3xTg-ADtauPETOther Journals

We would like to invite you to vote for your own “top ten” among 300 highly cited and most influential papers relevant to Alzheimer’s disease research.

LATEST JAD EDITORS’ BLOG
Is propagation of tau conformational change a "prion-like" mechanism?
Extracellular Tau Spreading
by Jesus Ávila, MD

The Alzheimer’s Funding Analyzer (AFA) showcases funding distribution from 42 countries and allows the end-user to conduct line-of-investigation queries to look for funding trends.

The Journal of Alzheimer's Disease

is an international multidisciplinary journal to facilitate progress in understanding the etiology, pathogenesis, epidemiology, genetics, behavior, treatment and psychology of Alzheimer's disease.

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Intracerebroventricular injection of Aβ1-42 in mice causes the death of pyramidal cells (arrows) in hippocampal CA1 regions, which was reduced by simvastatin at 20mg/kg, but aggravated at 80mg/kg. Scale bar=50 μm. From Jin et al., JAD50(2).

Chronic sleep deprivation (SD) for 2 months increases senile plaque burden in adult AβPPswe/PS1ΔE9 transgenic (TG) mice. Immunofluorescence staining of 6E10 showed the senile plaques (arrowhead). From Qiu et al., JAD50(3).

Immunofluorescence staining was conducted to locate p-STAT3 (green) in cytosol and nucleus. The nuclei were stained with DAPI (blue). Pre-treatment with gx-50 significantly increased the level of p-STAT3 in nucleus. Scale bar, 50 μm. From Shi et al., JAD50(3).

Decreased fractional anisotropy in subcortical vascular cognitive impairment patients compared to healthy subjects for 14 major fiber tracts. Red bands indicate significant differences (corrected through the cluster-based statistics). From Jung and Han et al., JAD50(4).

Hippocampal CA3 region of a 3xTg-AD mouse with cells showing Amyloid-beta (green) and Arc protein (red) immunoreactivity. Note the sparse presence of the two proteins and the high degree of co-expression (asterisks). From Morin et al., JAD51(1).

Tau in stromal nerve fascicles and simple cuboidal epithelial cells in human submandibular gland. From Dugger et al., JAD51(2).

18F-Flutemetamol, 18F-AV-1451, 18F-FDG PET and t1-mprage MRI images of a patient with Presenilin-1 mutation and early onset dementia. Color bars showing SUV ranges. Taken from Smith et al., JAD 51(2).

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Journal of Huntington's Disease
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Welcome to Journal of Alzheimer's Disease

Journal Ranking

Journal Ranking

The Journal of Alzheimer's Disease is an international multidisciplinary journal to facilitate progress in understanding the etiology, pathogenesis, epidemiology, genetics, behavior, treatment and psychology of Alzheimer's disease. The journal publishes research reports, reviews, short communications, hypotheses, ethics reviews, book reviews, and letters-to-the-editor. The journal is dedicated to providing an open forum for original research that will expedite our fundamental understanding of Alzheimer's disease.

JAD has an Impact Factor of 4.151 (2015 Journal Citation Reports), an h-index of 74 (Web of Science), an h-index of 70 (SCImago Journal Rank indicator), an h5-index of 59 (Google Scholar), and an h5-median of 78 (Google Scholar).

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