Biblio
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Author Title Type [ Year] Filters: First Letter Of Title is P [Clear All Filters]
“The Proactive Self in Space: How Egocentric and Allocentric Spatial Impairments Contribute to Anosognosia in Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 3, pp. 881-892, 2017.
, “proBDNF Accelerates Brain Amyloid-β Deposition and Learning and Memory Impairment in APPswePS1dE9 Transgenic Mice.”, J Alzheimers Dis, vol. 59, no. 3, pp. 941-949, 2017.
, “Prodromal Dementia with Lewy Bodies and Prodromal Alzheimer's Disease: A Comparison of the Cognitive and Clinical Profiles.”, J Alzheimers Dis, vol. 58, no. 2, pp. 463-470, 2017.
, “The Progression of Alzheimer's Disease: Are Fast Decliners Really Fast? A Four-Year Follow-Up.”, J Alzheimers Dis, vol. 57, no. 3, pp. 775-786, 2017.
, “A Prospective Study on the Association between Uric Acid and Cognitive Function among Middle-Aged and Older Chinese.”, J Alzheimers Dis, vol. 58, no. 1, pp. 79-86, 2017.
, “Protein Expression of BACE1 is Downregulated by Donepezil in Alzheimer's Disease Platelets.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1445-1451, 2017.
, “Protein Phosphorylation is a Key Mechanism in Alzheimer's Disease.”, J Alzheimers Dis, vol. 58, no. 4, pp. 953-978, 2017.
, “Proteomics Analysis of Blood Serums from Alzheimer's Disease Patients Using iTRAQ Labeling Technology.”, J Alzheimers Dis, vol. 56, no. 1, pp. 361-378, 2017.
, “PSD-93 Attenuates Amyloid-β-Mediated Cognitive Dysfunction by Promoting the Catabolism of Amyloid-β.”, J Alzheimers Dis, vol. 59, no. 3, pp. 913-927, 2017.
, “Psychosocial Risk and Protective Factors and Incident Mild Cognitive Impairment and Dementia in Community Dwelling Elderly: Findings from the Singapore Longitudinal Ageing Study.”, J Alzheimers Dis, vol. 57, no. 2, pp. 603-611, 2017.
, “Psychotic Symptoms Associated with Poor Renal Function in Mild Cognitive Impairment and Dementias.”, J Alzheimers Dis, vol. 58, no. 1, pp. 243-252, 2017.
, “Psychotropic Polypharmacy in Patients with Dementia: Prevalence and Predictors.”, J Alzheimers Dis, vol. 56, no. 2, pp. 707-716, 2017.
, “Pupillary Responses as a Biomarker of Early Risk for Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 4, pp. 1419-1428, 2017.
, “Pain Assessment in Elderly with Behavioral and Psychological Symptoms of Dementia.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1217-25, 2016.
, “Paired Studies Comparing Clinical Profiles of Lewy Body Dementia with Alzheimer's and Parkinson's Diseases.”, J Alzheimers Dis, vol. 54, no. 3, pp. 995-1004, 2016.
, “Palmomental Reflex a Relevant Sign in Early Alzheimer's Disease Diagnosis?”, J Alzheimers Dis, vol. 49, no. 4, pp. 1135-41, 2016.
, “Parkinsonism Differentiates Idiopathic Normal Pressure Hydrocephalus from Its Mimics.”, J Alzheimers Dis, vol. 54, no. 1, pp. 123-7, 2016.
, “Partial Amelioration of Synaptic and Cognitive Deficits by Inhibiting Cofilin Dephosphorylation in an Animal Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1419-32, 2016.
, “A Pathway Based Classification Method for Analyzing Gene Expression for Alzheimer's Disease Diagnosis.”, J Alzheimers Dis, vol. 49, no. 3, pp. 659-69, 2016.
, “Patients that have Undergone Hemodialysis Exhibit Lower Amyloid Deposition in the Brain: Evidence Supporting a Therapeutic Strategy for Alzheimer's Disease by Removal of Blood Amyloid.”, J Alzheimers Dis, vol. 51, no. 4, pp. 997-1002, 2016.
, “Patients with Amyloid-Negative Mild Cognitive Impairment have Cortical Hypometabolism but the Hippocampus is Preserved.”, J Alzheimers Dis, vol. 53, no. 2, pp. 651-60, 2016.
, “Patients with Mild Alzheimer's Disease Fail When Using Their Working Memory: Evidence from the Eye Tracking Technique.”, J Alzheimers Dis, vol. 50, no. 3, pp. 827-38, 2016.
, “Patterns of Brain Iron Accumulation in Vascular Dementia and Alzheimer's Dementia Using Quantitative Susceptibility Mapping Imaging.”, J Alzheimers Dis, vol. 51, no. 3, pp. 737-45, 2016.
, “Pauses During Autobiographical Discourse Reflect Episodic Memory Processes in Early Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 3, pp. 687-98, 2016.
, “PDE5 Exists in Human Neurons and is a Viable Therapeutic Target for Neurologic Disease.”, J Alzheimers Dis, vol. 52, no. 1, pp. 295-302, 2016.
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