Biblio
“Longitudinal Changes in Functional Brain Connectivity Predicts Conversion to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 377-89, 2016.
, “Longitudinal Memory Profiles in Behavioral-Variant Frontotemporal Dementia and Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 3, pp. 775-82, 2016.
, “Longitudinal Protein Changes in Blood Plasma Associated with the Rate of Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1105-14, 2016.
, “Looking for Neuroimaging Markers in Frontotemporal Lobar Degeneration Clinical Trials: A Multi-Voxel Pattern Analysis Study in Granulin Disease.”, J Alzheimers Dis, vol. 51, no. 1, pp. 249-62, 2016.
, “Low Plasma Cholinesterase Activities are Associated with Deficits in Spatial Orientation, Reduced Ability to Perform Basic Activities of Daily Living, and Low Body Mass Index in Patients with Progressed Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 3, pp. 801-13, 2016.
, “Low Prevalence of Mixed Dementia in a Cohort of 2,000 Elderly Patients in a Memory Clinic Setting.”, J Alzheimers Dis, vol. 50, no. 3, pp. 797-806, 2016.
, “Lower Prevalence of Alzheimer's Disease among Tibetans: Association with Religious and Genetic Factors.”, J Alzheimers Dis, vol. 50, no. 3, pp. 659-67, 2016.
, “Maintenance of Cognitive Performance and Mood for Individuals with Alzheimer's Disease Following Consumption of a Nutraceutical Formulation: A One-Year, Open-Label Study.”, J Alzheimers Dis, vol. 51, no. 4, pp. 991-5, 2016.
, “MAPT H1 Haplotype is Associated with Late-Onset Alzheimer's Disease Risk in APOEɛ4 Noncarriers: Results from the Dementia Genetics Spanish Consortium.”, J Alzheimers Dis, vol. 49, no. 2, pp. 343-52, 2016.
, “Measuring Information Processing Speed in Mild Cognitive Impairment: Clinical Versus Research Dichotomy.”, J Alzheimers Dis, vol. 51, no. 1, pp. 263-75, 2016.
, “Memory for Public Events in Mild Cognitive Impairment and Alzheimer's Disease: The Importance of Rehearsal.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1023-33, 2016.
, “A Meta-Analysis of Alzheimer's Disease Incidence and Prevalence Comparing African-Americans and Caucasians.”, J Alzheimers Dis, vol. 50, no. 1, pp. 71-6, 2016.
, “Metabolic Syndrome and Mild Cognitive Impairment: A Case-Control Study among Elderly in a Shanghai Suburb.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1175-82, 2016.
, “Metformin Facilitates Amyloid-β Generation by β- and γ-Secretases via Autophagy Activation.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1197-208, 2016.
, “Metformin in Amnestic Mild Cognitive Impairment: Results of a Pilot Randomized Placebo Controlled Clinical Trial.”, J Alzheimers Dis, vol. 51, no. 2, pp. 501-14, 2016.
, “Mild Cognitive Impairment and Susceptibility to Scams in Old Age.”, J Alzheimers Dis, vol. 49, no. 3, pp. 845-51, 2016.
, “Mindfulness in the Maintenance of Cognitive Capacities in Alzheimer's Disease: A Randomized Clinical Trial.”, J Alzheimers Dis, vol. 50, no. 1, pp. 217-32, 2016.
, “miR-302 Attenuates Amyloid-β-Induced Neurotoxicity through Activation of Akt Signaling.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1083-98, 2016.
, “A Mitochondrial Role of SV2a Protein in Aging and Alzheimer's Disease: Studies with Levetiracetam.”, J Alzheimers Dis, vol. 50, no. 1, pp. 201-15, 2016.
, “Moderate-to-High Intensity Physical Exercise in Patients with Alzheimer's Disease: A Randomized Controlled Trial.”, J Alzheimers Dis, vol. 50, no. 2, pp. 443-53, 2016.
, “Molecular markers of amnestic mild cognitive impairment among Mexican Americans.”, J Alzheimers Dis, vol. 49, no. 1, pp. 221-8, 2016.
, “Multimodal Magnetic Resonance Imaging in Alzheimer's Disease Patients at Prodromal Stage.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1035-50, 2016.
, “Naphthoquinone-Tryptophan Hybrid Inhibits Aggregation of the Tau-Derived Peptide PHF6 and Reduces Neurotoxicity.”, J Alzheimers Dis, vol. 51, no. 1, pp. 165-78, 2016.
, “Neurophysiological assessment of Alzheimer's disease individuals by a single electroencephalographic marker.”, J Alzheimers Dis, vol. 49, no. 1, pp. 159-77, 2016.
, “Non-Verbal Episodic Memory Deficits in Primary Progressive Aphasias are Highly Predictive of Underlying Amyloid Pathology.”, J Alzheimers Dis, vol. 51, no. 2, pp. 367-76, 2016.
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