Biblio

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Journal Article
K. Krishna, Behnisch, T., and Sajikumar, S., “Inhibition of Histone Deacetylase 3 Restores Amyloid-β Oligomer-Induced Plasticity Deficit in Hippocampal CA1 Pyramidal Neurons.”, J Alzheimers Dis, vol. 51, no. 3, pp. 783-91, 2016.
K. Rajah Kumaran, Yunusa, S., Perimal, E., Wahab, H., Müller, C. P., and Hassan, Z., “Insights into the Pathophysiology of Alzheimer's Disease and Potential Therapeutic Targets: A Current Perspective.”, J Alzheimers Dis, vol. 91, no. 2, pp. 507-530, 2023.
I. Frentz, van Arendonk, J., Leeuwis, A. E., Vernooij, M. W., van der Flier, W. M., Bos, D., De Deyn, P. Paul, Wolters, F. J., and M Ikram, A., “Interaction Between Arteriosclerosis and Amyloid-β on Cognitive Function.”, J Alzheimers Dis, vol. 97, no. 2, pp. 953-961, 2024.
H. Xu, Perreau, V. M., Dent, K. A., Bush, A. I., Finkelstein, D. I., and Adlard, P. A., “Iron Regulates Apolipoprotein E Expression and Secretion in Neurons and Astrocytes.”, J Alzheimers Dis, vol. 51, no. 2, pp. 471-87, 2016.
A. Koppara, Wolfsgruber, S., Kleineidam, L., Schmidtke, K., Frölich, L., Kurz, A., Schulz, S., Hampel, H., Heuser, I., Peters, O., Reischies, F. M., Jahn, H., Luckhaus, C., Hüll, M., Gertz, H. - J., Schröder, J., Pantel, J., Rienhoff, O., Rüther, E., Henn, F., Wiltfang, J., Maier, W., Jessen, F., Kornhuber, J., and Wagner, M., “The Latent Dementia Phenotype δ is Associated with Cerebrospinal Fluid Biomarkers of Alzheimer's Disease and Predicts Conversion to Dementia in Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 49, no. 2, pp. 547-60, 2016.
L. Yu, Liu, Y., Jin, Y., Cao, X., Chen, J., Jin, J., Gu, Y., Bao, X., Ren, Z., Xu, Y., and Zhu, X., “Lentivirus-Mediated HDAC3 Inhibition Attenuates Oxidative Stress in APPswe/PS1dE9 Mice.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1411-1424, 2018.
J. D. 'Arcy Rudge, “The Lipid Invasion Model: Growing Evidence for This New Explanation of Alzheimer's Disease.”, J Alzheimers Dis, vol. 94, no. 2, pp. 457-470, 2023.
G. Lombardi, Berti, V., Tedde, A., Bagnoli, S., Piaceri, I., Polito, C., Lucidi, G., Ferrari, C., Ginestroni, A., Moretti, M., Pupi, A., Nacmias, B., and Sorbi, S., “Low Florbetapir PET Uptake and Normal Aβ1-42 Cerebrospinal Fluid in an APP Ala713Thr Mutation Carrier.”, J Alzheimers Dis, vol. 57, no. 3, pp. 697-703, 2017.
E. C. Pinnock, Jovanovic, K., Pinto, M. G., Ferreira, E., Dias, B. Da Costa, Penny, C., Knackmuss, S., Reusch, U., Little, M., Schatzl, H. M., and Weiss, S. F. T., “LRP/LR Antibody Mediated Rescuing of Amyloid-β-Induced Cytotoxicity is Dependent on PrPc in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 3, pp. 645-57, 2016.
D. A. Gimbel, Nygaard, H. B., Coffey, E. E., Gunther, E. C., Laurén, J., Gimbel, Z. A., and Strittmatter, S. M., “Memory impairment in transgenic Alzheimer mice requires cellular prion protein.”, J Neurosci, vol. 30, no. 18, pp. 6367-74, 2010.
S. - S. Wang, Jia, J., and Wang, Z., “Mesenchymal Stem Cell-Derived Extracellular Vesicles Suppresses iNOS Expression and Ameliorates Neural Impairment in Alzheimer's Disease Mice.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1005-1013, 2018.
S. Min Son, Shin, H. - J., Byun, J., Kook, S. Young, Moon, M., Chang, Y. Jin, and Mook-Jung, I., “Metformin Facilitates Amyloid-β Generation by β- and γ-Secretases via Autophagy Activation.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1197-208, 2016.
J. A. Luchsinger, Perez, T., Chang, H., Mehta, P., Steffener, J., Pradabhan, G., Ichise, M., Manly, J., Devanand, D. P., and Bagiella, E., “Metformin in Amnestic Mild Cognitive Impairment: Results of a Pilot Randomized Placebo Controlled Clinical Trial.”, J Alzheimers Dis, vol. 51, no. 2, pp. 501-14, 2016.
H. - H. Li, Lin, S. - L., Huang, C. - N., Lu, F. - J., Chiu, P. - Y., Huang, W. - N., Lai, T. - J., and Lin, C. - L., “miR-302 Attenuates Amyloid-β-Induced Neurotoxicity through Activation of Akt Signaling.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1083-98, 2016.
E. M. Elzayat, Shahien, S. A., El-Sherif, A. A., and Hosney, M., “miRNAs and Stem Cells as Promising Diagnostic and Therapeutic Targets for Alzheimer's Disease.”, J Alzheimers Dis, vol. 94, no. s1, pp. S203-S225, 2023.
T. Kondo, Asai, M., Tsukita, K., Kutoku, Y., Ohsawa, Y., Sunada, Y., Imamura, K., Egawa, N., Yahata, N., Okita, K., Takahashi, K., Asaka, I., Aoi, T., Watanabe, A., Watanabe, K., Kadoya, C., Nakano, R., Watanabe, D., Maruyama, K., Hori, O., Hibino, S., Choshi, T., Nakahata, T., Hioki, H., Kaneko, T., Naitoh, M., Yoshikawa, K., Yamawaki, S., Suzuki, S., Hata, R., Ueno, S. - I., Seki, T., Kobayashi, K., Toda, T., Murakami, K., Irie, K., Klein, W. L., Mori, H., Asada, T., Takahashi, R., Iwata, N., Yamanaka, S., and Inoue, H., “Modeling Alzheimer's disease with iPSCs reveals stress phenotypes associated with intracellular Aβ and differential drug responsiveness.”, Cell Stem Cell, vol. 12, no. 4, pp. 487-96, 2013.
Y. Huang, Shen, W., Su, J., Cheng, B., Li, D., Liu, G., Zhou, W. - X., and Zhang, Y. - X., “Modulating the Balance of Synaptic and Extrasynaptic NMDA Receptors Shows Positive Effects against Amyloid-β-Induced Neurotoxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 885-897, 2017.
A. Ezra, Rabinovich-Nikitin, I., Rabinovich-Toidman, P., and Solomon, B., “Multifunctional Effect of Human Serum Albumin Reduces Alzheimer's Disease Related Pathologies in the 3xTg Mouse Model.”, J Alzheimers Dis, vol. 50, no. 1, pp. 175-88, 2016.
M. González-Ramírez, Gavilán, J., Silva-Grecchi, T., Cajas-Madriaga, D., Triviño, S., Becerra, J., Saez-Orellana, F., Pérez, C., and Fuentealba, J., “A Natural Benzofuran from the Patagonic Aleurodiscus vitellinus Fungus has Potent Neuroprotective Properties on a Cellular Model of Amyloid-β Peptide Toxicity.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1463-1475, 2018.
R. Zhang, Song, Y., and Su, X., “Necroptosis and Alzheimer's Disease: Pathogenic Mechanisms and Therapeutic Opportunities.”, J Alzheimers Dis, vol. 94, no. s1, pp. S367-S386, 2023.
X. Chen, Jiang, S., Wang, R., Bao, X., and Li, Y., “Neural Stem Cells in the Treatment of Alzheimer's Disease: Current Status, Challenges, and Future Prospects.”, J Alzheimers Dis, vol. 94, no. s1, pp. S173-S186, 2023.
S. V. Ovsepian and O'Leary, V. B., “Neuronal activity and amyloid plaque pathology: an update.”, J Alzheimers Dis, vol. 49, no. 1, pp. 13-9, 2016.
A. W. Bero, Yan, P., Roh, J. Hoon, Cirrito, J. R., Stewart, F. R., Raichle, M. E., Lee, J. - M., and Holtzman, D. M., “Neuronal activity regulates the regional vulnerability to amyloid-β deposition.”, Nat Neurosci, vol. 14, no. 6, pp. 750-6, 2011.
C. Castillo, Bravo-Arrepol, G., Wendt, A., Saez-Orellana, F., Millar, C., Burgos, C. F., Gavilán, J., Pacheco, C., Ahumada-Rudolph, R., Napiórkowska, M., Pérez, C., Becerra, J., Fuentealba, J., and Cabrera-Pardo, J. R., “Neuroprotective Properties of Eudesmin on a Cellular Model of Amyloid-β Peptide Toxicity.”, J Alzheimers Dis, vol. 94, no. s1, pp. S97-S108, 2023.
M. Van Zeller, Dias, D., Sebastião, A. M., and Valente, C. A., “NLRP3 Inflammasome: A Starring Role in Amyloid-β- and Tau-Driven Pathological Events in Alzheimer's Disease.”, J Alzheimers Dis, vol. 83, no. 3, pp. 939-961, 2021.

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